Following infection with COVID-19, several individuals have developed diabetic symptoms. Scientists are now wondering whether COVID-19 may cause diabetes. The coronavirus may be causing the pancreas to self-destruct, according to preliminary results. More business articles may be found on Insider’s business page.
According to a recent study being conducted by experts, the coronavirus may be damaging critical cells in the pancreas, resulting in diabetes.
Diabetes After COVID-19 New Study Indicates
COVID-19 and diabetes have a complicated connection that scientists are still trying to figure out.
However, as the epidemic proceeded, an increasing number of reports indicated that individuals infected with COVID-19 were experiencing their first signs of diabetes. It’s too early to tell whether the condition will last.
Francesco Rubino, head of metabolic surgery at King’s College London, told Insider that there is a connection; some process helps the illnesses feed one another. The issue is whether this virus may induce new-onset diabetes.
Diabetes is an overabundance of blood sugar caused by the body’s inability to produce enough insulin, the hormone that regulates blood sugar, or its resistance to it. One hypothesis suggested that the body mistook pancreatic cells for coronavirus and attempted to kill them. The scientists hypothesized that this would impair insulin delivery and result in diabetes.
However, research indicates that something more is going on: the virus may be changing the pancreas, causing it to self-destruct. On April 1, 2020, a woman wearing a facemask wandered through San Francisco. The very high amounts of blood sugar that individuals generate following COVID-19 are a characteristic of diabetes.
To combat this, Shiubing Chen, a researcher at Weill Cornell Medicine’s Department of Surgery, told Insider that large amounts of insulin are required. Chen and her colleagues used COVID-19 to examine postmortem samples from five donors to figure out what was happening in the pancreas. They also infected cells obtained from healthy human pancreases in the lab with the coronavirus.
On August 3, their research was published in the peer-reviewed journal Cell Metabolism. The researchers discovered that following infection, the pancreas’ insulin-producing cells began to behave oddly. They stopped producing insulin and began producing glucagon, a hormone that has the opposite effect.
In addition, the cells began producing trypsin, a digesting enzyme, and chemokines, a kind of chemical that informs immune system cells that they are ill and should be eliminated. We do not know yet whether this impact is strong enough to induce diabetes to develop where there was none before, Chen added.
A woman holds a dosage of COVID-19 vaccination in her hand. ReutersCOVID-19’s interaction with diabetes is unknown. According to Rubino, a metabolic surgery specialist, the coronavirus may alter beta cell activity.
However, he said, there may be other factors at play. It is conceivable, for example, that some of the individuals had diabetes but were unaware of it before contracting COVID-19.
Rubino has helped create a register for new-onset diabetes patients, which he believes will shed light on the problem. According to Rubino and Chen, it’s unclear how long diabetic symptoms would persist following infection. Both experts agreed that being vaccinated is the best way to prevent contracting COVID-19.